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Metabolic influences on reward circuits That the mesolimbic dopaminergic pathway is involved in the reinforcing and addictive properties of drugs of abuse has been well documented ever since Roberts, Corcoran, and Fibiger reported that catecholaminergic lesions of the nucleus accumbens reduced self-administration of cocaine in a rodent model. Although the underlying mechanisms for this phenomenon have not been completely elucidated, the involvement of intrinsic CNS circuitry and transmitters has been identified in food reward behavior and function and suggests, indeed, multiple links between feeding, nutritional status, and reward circuitry. Subsequent research, including that from the laboratory of Ann E.

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  1. Studies in the s demonstrated that insulin and leptin can decrease food reward in rats assessed by two different tasks:

  2. Kelley emphasized the insight that basic neuroscience research into the mechanisms of reward Kelley and Berridge, and learning and memory Kelley, provided in terms of understanding the processes and neural substrates that regulate adaptive behavior, and which are often driven in maladaptive ways by exposure to drugs of abuse and to the current food environment.

  3. Obesity is a complex metabolic syndrome that is characterized by energy dyshomeostasis and involves not only the brain, but also basic biochemical reactions within liver, fat, and muscle tissue.